…Prevention of recurrence after a healed venous ulcer. After deep vein thrombosis with post thrombotic syndrome. Differential diagnosis Feature Venous ulcer Arterial ulcer Diabetic foot…

…and a small lipid core, the pathophysiology of Acute Coronary Syndromes (ACS) is driven by the sudden destabilization of these lesions, a process known as…

…progressive, and highly complex lipid driven inflammatory disease of the arterial intima that serves as the fundamental pathophysiological substrate for acute coronary syndromes (ACS), including…

…Monitor coagulation. Cyclosporine, tacrolimus: NSAIDs may increase the nephrotoxic effects of cyclosporine and tacrolimus. Monitor kidney function. Diuretics and antihypertensives: NSAIDs may reduce the efficacy…

…lipid laden foamy macrophages accumulate within the neointima. It may be accompanied by necrotic core formation and calcification. Neoatherosclerosis can contribute to both ISR and…

…investigation of restenosis and stent thrombosis. Intravascular imaging may also clarify the mechanism of an acute coronary syndrome (ACS), particularly when angiography shows no significant…

…may be observed after large myocardial infarctions, in which the large necrotic area may cause nonspecific conduction disturbances. Such conduction disturbances may also be superimposed…

…that can trigger or sustain arrhythmias, such as necrotic tissue after myocardial infarction (refer to Mechanisms of cardiac arrhythmias). It is often difficult to clarify…

…or underlying channelopathies (e.g., Torsades de Pointes in Long QT Syndrome) [1] . Underlying Etiologies The etiology of VT is broadly dichotomized into structural heart…

…cardiac dysfunction, haemodynamic instability, anaemia and other nephrotoxic exposures may contribute to tubular injury, making attribution to contrast alone difficult. In patients with normal renal…