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Delirium in older adults: quick reference

…whether the condition is new or clearly worse than baseline. Contact relatives, home care services, the residential care home or the previous care provider. 5…

ST-Elevation Myocardial Infarction (STEMI): Pathophysiology, Diagnosis, and Management

…progressive, and highly complex lipid driven inflammatory disease of the arterial intima that serves as the fundamental pathophysiological substrate for acute coronary syndromes (ACS), including…

Targeted temperature management after cardiac arrest

…has therefore not been abandoned, only redefined: continuous core temperature measurement and a target value that must never be exceeded, during the first 72 hours…

ST-Elevation Myocardial Infarction: Diagnosis and Electrocardiographic Criteria

…coronary occlusion. Plaque rupture typically involves a thin, collagen poor fibrous cap overlying a large lipid core with many macrophages, resulting in a red, fibrin…

Vulnerable Plaque and Mechanisms of Acute Coronary Occlusion

…vulnerable plaque” describes an atherosclerotic lesion thought to be susceptible to disruption and thrombosis. Typical features include a lipid rich core, a thin fibrous cap…

Non-ST Elevation Acute Myocardial Infarction (NSTEMI) and Unstable Angina

…of atherosclerotic plaques [1] . While stable plaques are characterized by a thick fibrous cap and a small lipid core, the pathophysiology of Acute Coronary Syndromes…

Atherosclerosis: Pathogenesis from Fatty Streak to Plaque Rupture

…and extracellular matrix beneath the endothelium. Plaques that are particularly prone to rupture characteristically contain: a large lipid rich core; abundant foam cells and macrophages…

In-Stent Restenosis: Mechanisms and Treatment

…lipid laden foamy macrophages accumulate within the neointima. It may be accompanied by necrotic core formation and calcification. Neoatherosclerosis can contribute to both ISR and…

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