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Part 1: Basic Cardiac Electrophysiology
These lessons will introduce you to the key concepts of electrocardiology and ECG interpretation. Basic cardiac anatomy, cellular function, features of contractile and conduction cells, action potential, automaticity and the electrical conduction system is discussed in detail.
Part 2: Recognizing the Normal ECG
These lessons will teach you all about the normal ECG, normal variants and diagnosis of pathological findings. A total of 8 lessons will guide you through all the waves, intervals and durations, including the PR interval, QRS complex, ST segment, J point, J 60 point, T-wave, QT interval and the electrical axis of the heart. Test your skills with the ECG quiz after the lectures.
Test your skills
Part 3: ECG test 1
The first ECG test is a quiz (multiple choice) with detailed answers.
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An introduction to ECG interpretation, cardiac physiology and concepts of electrocardiology
The nuts and bolts of cardiac electrophysiology.
Learn about lead theory, ECG lead systems, chest (precordial) leads, limb leads, presentation of leads and alternative lead systems.
Learn why the standard presentation of the 12-lead ECG is inferior to the Cabrera format with an inverted lead aVR.
This is perhaps the most important chapter throughout this book. You will learn how to recognize the normal ECG and identify pathological ECG changes.
Learn how to interpret the ECG efficienty using a systematic approach.
Part 1: Basic Arrhythmology
Learn about the three mechanisms causing cardiac arrhythmias: re-entry (reentry), increased automaticity and triggered activity.
Aberrant ventricular conduction is not an arrhythmia per se but may frequently mimic arrhythmias.
Learn how to diagnose premature atrial beats (or atrial extrasystoles, premature atrial contractions) and differentiate them from other diagnoses.
Learn how to recognize premature ventricular beats (or ventricular extrasystoles, premature ventricular contractions) and differentiate them from other diagnoses.
Learn the ECG criteria, physiology and characteristics of sinus rhythm, which is the normal rhythm of the heart.
Learn ECG characteristics and implications of sinus arrhythmia in various patient populations.
Part 2: Bradyarrhythmia (bradycardia)
Note that management (treatment) of bradycardia is discussed in the final chapter (Sinus Node Dysfunction & Sick Sinus Syndrome).
Learn about causes and management of sinus bradycardia.
Chronotropic incompetence is the inability to increase the heart rate adequately, learn about it in this chapter.
Temporary malfunction in the sinoatrial node may lead to these two conditions which must be diagnosed whenever present.
Learn about SA blocks and how to diagnose and manage them.
SND and SSS are common clinical conditions. In this chapter you will learn about these conditions and you will also learn how to manage bradycardias regardless of the underlying cause.
Part 3: Supraventricular Tachyarrhythmias (tachycardia)
- Sinus Tachycardia, Inapproproate Sinus Tachycardia, Sinoatrial Re-entry Tachycardia & High Resting Heart Rate
These are some of the most common and often puzzling conditions in clinical practice. Learn all about them in this chapter.
Learn classification, causes, diagnostic criteria and management of atrial fibrillation, which is the most common clinically significant tachyarrhythmia.
Learn classification, causes, diagnostic criteria and management of atrial flutter.
There are three types of atrial rhythms: ectopica atrial tachycardia, atrial rhythm and multifocal atrial tachycardia. Learn about them in this chapter.
AVNRT is both common and difficult to recognize, which is why this chapter provides an in-depth discussion on causes, diagnosis, ECG manifestation and management.
The prevalence of pre-excitation is 1 per 1000 individuals and the condition may cause frequent arrhythmias. Learn all about pre-excitation and WPW syndrome in this chapter.
The atrioventricular (AV) node may also participate in arrhythmias, which is discussed in this chapter.
Part 4: Ventricular Tachyarrhythmias (tachycardia)
Occasionally rhythms may arise in the ventricular myocardium. Slower ventricular rhythms are either simply ventricular rhythm or accelerated (idioventricular) rhythm, both of which are discussed in this chapter.
The most common of the potentially fatal arrhythmias. Ventricular tachycardia (VT) must be recognized on the ECG and management must be evidence based in order to save lives.
LQTS manifests with an abnormally long QT interval and it may precipitate lethal arrhythmias. Learn all about LQTS in this chapter.
Ventricular fibrillation is discussed in this chapter.
Cardiac devices, particularly pacemakers, may also cause arrhythmias. It is important to be able to differentiate PMT from true ventricular tachycardia and other tachyarrhythmias.
Part 5: Clinical Management of Tachyarrhythmias (tachycardia)
Diagnosing and managing various tachyarrhythmias may be difficult. This chapter provides an in-depth guide to clinical reasoning, differential diagnostics and management of tachyarrhtyhmias (tachycardias). General aspects as well as details on narrow complex tachycardia (NCT) and wide complex tachycardia (WCT) are covered.
Part 1. Principles of Ischemic Heart Disease (Coronary Artery Disease)
Coronary artery disease is the number one killer worldwide. This chapter will introduce you to basic concepts of ischemic heart disease.
The acute complication of coronary artery disease occurs when an athersclerotic plaque ruptures. This results in atherothrombosis which leads to an acute coronary syndrome. This chapter will introduce you to pathophysiology, classification and clinical implications of acute coronary syndromes.
Optimal handling of ECG increases survival in patients with chest pain (acute coronary syndromes). This chapter discusses the essentials of ECG in the management of chest pain patients.
Learn how acute myocardial infarction is diagnosed and the difference between ST elevation myocardial infarction (STEMI), Non-ST elevation myocardial infarction (NSTEMI) and unstable angina (UA).
Understanding how the myocardium reacts during ischemia is essential to understanding ECG changes in ischemic heart disease. This chapter will discuss principles governing myocardial oxygen consumption as well as myocardial reaction during ischemia. You should also read the complimentary chapter: The Ischemic Cascade during Exercise Testing.
The natural course of acute myocardial infarction is discussed in this chapter.
Learn why myocardial infarction is equal to left ventricular infarction.
Part 2: The ECG in Ischemia and Infarction
Acute ischemia causes ST-T changes. This is a difficult topic which is discussed in detail in this and the subsequent chapters.
Learn all about ST segment depressions. This is an in-depth guide to causes and implications of various ST segment depressions, including those caused by acute myocardial ischemia / infarction.
Learn all about ST segment elevations. This is an in-depth guide to causes and implications of various ST segment elevations, including those caused by acute myocardial ischemia / infarction.
Learn about clinical syndromes that do not directly manifest with ST segment elevations but must be managed as STEMI in order to improve survival.
T-wave changes are frequently misunderstood, particularly in the setting of ischemia and infarction. This chapter provides a detailed discussion on the causes and implications of various T-wave changes (flat T-waves, inverted [negative] T-waves and hyperacute T-waves).
Learn how to differentiate various Q-wave changes, including normal Q-waves, normalvariants and pathological Q-waves. Pathological R-waves are also discussed.
Learn why left bundle branch block poses a special challenge in patients with acute coronary syndromes.
Ischemia may cause additional ECG changes which are discussed in this chapter.
- Conduction Defects and Blocks in Ischemia and Infarction 17. Reperfusion assessment using 12-lead ECG 18. Localization of Ischemic and Infarct Area Using 12-lead ECG
In this chapter you will learn how to use the 12-lead ECG to localize the myocardial ischemia/infarction.
Part 3: Management of Acute Coronary Syndromes (ACS)
In these comprehensive chapters you will learn how to manage patients with chest pain, patients with STEMI and patients with NSTEMI.
Part 1. Supraventricular Conduction Defects
- Overview of Atrioventricular (AV) Blocks
- First-degree AV block
- Second-degree AV block
- Third-degree AV block
- Management and Treatment of AV block
Part 2. Intraventricular Conduction Defects
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J waves occur in the J point, which is the terminal portion of the QRS complex. J waves vary in amplitude and shape and they may be caused by both benign and potentially life-threatening conditions. A clinical syndrome which manifests with abnormal J waves is referred to as a J wave syndrome. This chapter discusses general aspects of the J wave syndromes.
This chapter provides an in-depth discussion on the Brugada syndrome, which is a rather mysterious and potentially life-threatening genetic disorder.
Early repolarization is one of the most frequently misunderstood syndromes. It is common and associated with 3 to 5 times as great a risk of sudden cardiac death. Learn all about early repolarization in this chapter.
This chapter elucidates electrocardiographic aspects of acute myocarditis (percarditis, perimyocarditis).
Learn about cardiac tamponade and electrical alternans, and how the ECG may be used to diagnose tamponade.
Learn about takotsubo cardiomyopathy and why this condition frequently results in unnecessary activations of the catheterization laboratory.
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- Introduction to exercise stress testing (exercise ECG)
- Indications, contraindications and preparations for exercise (ECG) stress testing
- Exercise protocols (treadmill, cycle), evaluation and termination
- Exercise stress testing in special patient population
- Exercise physiology: from normal response to myocardial ischemia
- Evaluation of test results: ECG reaction and other parameters